By Jesús Avila
Content material: Preface; Animal versions on Alzheimer affliction; Animal versions for Amyloid/PS-1 Pathology; Transgenic mice overexpressing GSK-3beta as animal types of Alzheimer affliction; Reelin and Alzheimer affliction; Tauopathy versions; Animal versions for Parkinson disorder I; Animal versions for Parkinson affliction II; Neuroprotection in Parkinson sickness; Animal versions for ALS; Mouse types of Huntington's illness; Recesive Ataxias; Mouse versions in PrP-related illnesses; Mouse types of ischemia
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Additional info for Animal Models for Neurodegenerative Disease
APP23 mice exhibit rare Congo-red positive Ab plaques in the brain at six months, and Ab plaques increase in size and number in neocortex and hippocampus with advancing age. 59 APP17 mice express hAPP with the Swedish mutation, and APP22 mice express hAPP with the Swedish and London (V717I) mutations, both under control of the human Thy1 promoter. These two mouse lines express twofold more hAPP compared with that of endogenous mouse APP. APP17 mice do not show Ab deposition in the brain until 24 months of age.
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