By J. F. Theis, C. S. Newlon (auth.), Prof. Dr. Robert Brambl, Prof. Dr. George A. Marzluf (eds.)
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Additional resources for Biochemistry and Molecular Biology
Cell 5:263-269 Caddie MS, Calos MP (1994) Specific initiation at an origin of replieation from Schizosaccharomyces pombe. Mol Cell BioI14:1796-1805 Yeast Chromosome Replieation Campbell JL, Newlon CS (1991) Chromosomal DNA replication. In Broaeh JR, Pringle JR, Jones E (eds) The moleeular and eellular biology of the yeast Saccharomyces: genome dynamies, protein synthesis, and energeties. Cold Spring Harbor Lab Press, Cold Spring Harbor, pp 41-146 Celniker SE, Sweder K, Sriene F, Bailey JE, Campbell JL (1984) Deletion mutations affeeting autonomously replieating sequenee ARS1 of Saccharomyces cerevisiae.
Biochim Biophys Acta 1217:239-256 Rowley A, Cocker JH, Harwood 1, Diffley IFX (1995) Initiation complex assembly at budding yeast replication origins begins with the recognition of abipartite sequence by limiting amounts of the initiator, ORe. EMBO 114:2631-2641 Saffer LD, Miller OL Ir (1986) Electron microscopic study of Saccharomyces cerevisiae rDNA chromatin replication. Mol Cell Biol 6:1148-1157 Schild D, Byers B (1978) Meiotic effects of DNA-defective cell division cycle mutations of Saccharomyces cerevisiae.
RAR3 and RAR5 turned out to be previously isolated genes. RAR3 is the same as GALII I SPT13, and RAR5 is DST2 (Kipling et al. 1991). DST2 encodes DNA strand transfer pro tein ß, an activity presumably involved in recombination (Dykstra et al. 1991), and the gene has also been identified as an exoribonuclease and in a screen for genes that enhance the nuclear fusion defects of karl mutants of budding yeast (reviewed by Kearsey and Kipling 1991). How RAR5 affects replication is unclear, though the plasmid stabilization phenotype results from inactivation of the gene.