Central Pain Syndrome: Pathophysiology, diagnosis and by Sergio Canavero

By Sergio Canavero

Significant soreness Syndrome is a neurological situation brought on by harm particularly to the critical worried method - mind, brainstem, or spinal wire. this can be the single up to date ebook to be had at the medical features (including analysis and treatment) of CPS administration. The authors have built a really whole reference resource on critical discomfort, consisting of heritage fabric, pathophysiology, and diagnostic and healing info. A scientific secret for a hundred years with out potent treatment, this booklet turns the concept that of incurability of relevant soreness on its head supplying a rational method of remedy according to a rational thought.

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Extra resources for Central Pain Syndrome: Pathophysiology, diagnosis and management

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019) 12 spinal Garcia-Larrea et al. 5) 119 Weimar et al. (2002) Widar et al. 5 (40À96, median 74) 8 Bowsher (2001) Lampl et al. 7 (35À85, median 58) 16 CPSP group Kumral et al. 3 (continued) 31 Pathology Michel et al. (1990) Tasker et al. (1991) Kameyama (1976) Graff-Radford et al. (1985) Kawahara et al. (1986) Bogousslavsky et al. (1988) Mauguie`re and Desmet (1988) Leijon et al. (1989) and Boivie et al. 5%) 20 (74%) 1 (33%) 1 (25%) 4 (36%) CPSP Stroke (same goup of CPSP pts for 2 different studies.

The hemiface, one hand, one foot, a quadrant of the body, or the mouth and hand (the cheiro-oral syndrome), without a transition zone; the face and arm are most affected, and the leg least, reflecting greater cortical representation, but a hemiface singly is affected in roughly 10% of the cases. The pain may vary in site (‘‘wander’’), disappearing from one limb only to arise in another, and intense pains in the limbs may be found simultaneously with only paresthesias in the face, or vice versa (Garcin 1937; Riddoch 1938).

More recently, after the introduction of new imaging technologies (CT, MRI), the site and the volume of the lesions have been reported in papers dealing with thalamic stroke. In some papers the authors also reported the occurrence of CPSP, allowing the evaluation of the lesion volume in CPSP cases. Apparently, the volume of the lesion in patients with thalamic CPSP does not seem to differ from the expected volume in thalamic hemorrhage, nor between patients with somatosensory deficits with and without CPSP.

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