By David (ed.) Evered
Chapter 1 Chairman's creation (pages 1–3): B. C. Clarke
Chapter 2 model of crops to Soils Containing poisonous Metals ? A try out for Conceit (pages 4–19): A. D. Bradshaw
Chapter three version of vegetation to Mineral pressure in challenge Soils (pages 20–39): C. D. Foy
Chapter four Plant?Plant Interactions (pages 40–51): E. A. Bell
Chapter five variation of vegetation to Atmospheric toxins (pages 52–72): Thomas C. Hutchinson
Chapter 6 Evolution of Herbicide?Resistant Weeds (pages 73–99): J. Gressel
Chapter 7 Phytoalexins and ailment Resistance Mechanisms from a standpoint of Evolution and version (pages 100–118): Joseph Kuc
Chapter eight Insect?Plant Interactions: Endocrine Defences (pages 119–137): W. S. Bowers
Chapter nine Insect?Plant variations (pages 138–151): T. R. E. Southwood
Chapter 10 edition of bugs to pesticides (pages 152–166): R. M. Sawicki and that i. Denholm
Chapter eleven Biochemical Mechanisms of Resistance to pesticides (pages 167–189): Ernest Hodgson and Naoki Motoyama
Chapter 12 variation of Fungi to Fungitoxic Compounds (pages 190–203): S. G. Georgopoulos
Chapter thirteen Bacterial Resistance to Antibiotics (pages 204–218): Naomi Datta
Chapter 14 Evolutionary Relationships between Genes for Antibiotic Resistance (pages 219–232): Julian Davies and Gary Gary
Chapter 15 accent DNAs within the Bacterial Gene Pool: Playground for Coevolution (pages 233–252): Daniel L. Hartl, Daniel E. Dykhuizen and Douglas E. Berg
Chapter sixteen Chairman's remaining comments (pages 253–257): B. C. Clarke
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Additional info for Ciba Foundation Symposium 102 - Origins and Development of Adaptation
Nfectional compounds); the second class comprises compounds that are detectable only after infection (post-infectional compounds). Of the pre-infectional compounds, some (the prohibitins) are able, at their pre-infectional concentrations in the higher plant, to halt the in vivo multiplication of microorganisms. Others (the inhibitins) must increase in concentration after infection before they can exercise effective control over the invading pathogen. Of the post-infectional compounds, some (the post-inhibitins) are formed by the modification of pre-existing non-toxic precursors while the phytoalexins (dealt with elsewhere; see KuC, this volume) are synthesized de novo after infection.
Indian Society of Genetics and Plant Breeding, Indian Agricultural Research Institute, New Delhi 110012. India, vol 2, p 1241-1253 Vose PB 1982 Rationale of selection for specific nutritional characters in crop improvement with Phaseolus vulgaris L. as a case study. In: Saric MR (ed) Genetic specificity of mineral nutrition of plants. Serb Acad Sci Art. Scientific Assemblies, Dept Nat Math Sci Vol XI11 No 3, Belgrade, Yugoslavia, p 313-323 Weiss-MG 1943Inheritance and physiology of efficiency in iron-utilization in soybeans.
Manganese tolerance Manganese tolerance has been associated with the following factors: higher oxidizing powers of plant roots (or associated microorganisms); lower manganese absorption and translocation rates; trapping of excess manganese in non-metabolic centres; higher internal tolerance to excess manganese (possibly through a chelation4etoxification mechanism); and more efficient uptake and distribution of iron and silicon. Current evidence from our laboratory suggests that in genotypes of cotton, wheat, soybean and snapbean, tolerance to high manganese concentrations within the plant is more important than reduced manganese uptake in determining manganese tolerance (Foy 1973, 1983c, Foy et al 1978).